Cure Your Child with Food!
The Hidden Connection Between Nutrition and Childhood Ailments
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Cure Your Child with Food!
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Narrated by:
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Ann Marie Lee
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By:
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Kelly Dorfman
About this listen
Why treat your child with drugs when you can cure your child with nutrition? Grounded in cutting-edge science and filled with case studies that read like medical thrillers, this is a book for every parent whose child suffers from mood swings, stomachaches, ear infections, eczema, anxiety, tantrums, ADD/ADHD, picky eating, asthma, lack of growth, and a host of other physical, behavioral, and developmental problems.
Previously published as What's Eating Your Child? and now with a new chapter on the unexpected connection between gluten and insatiable appetite, Cure Your Child with Food shows parents how to uncover the clues behind their children's surprisingly nutrition-based health issues and implement simple treatments - immediately.
You'll discover how zinc deficiency can cause picky eating and affect growth; the panoply of problems caused by gluten and dairy; and how ear infections and mood disorders, such as anxiety and bipolar disorder, can be a sign of food intolerance. Plus, you'll learn how to get your child to sleep, soothe hyperactivity, and deal with reflux using simple nutritional strategies.
Dorfman, a nutritionist whose typical family arrives at her practice after seeing three or more specialists, gives parents the tools they need to become nutrition detectives; to recalibrate their children's diets through the easy E.A.T. program; and, finally, to get their children off drugs - antibiotics, laxatives, Prozac, Ritalin - and back to a natural state of well-being.
©2011, 2013 Kelly Dorfman (P)2013 TantorListeners also enjoyed...
What listeners say about Cure Your Child with Food!
Average customer ratingsReviews - Please select the tabs below to change the source of reviews.
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Overall3 out of 5 stars
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Performance5 out of 5 stars
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Story5 out of 5 stars
- Madeleine de Sarah
- 01-09-22
Some of the information wasn't correct
Should be noted that taking melatonin(which is a hormone) should only be done for a short period of time, since your body gets used and starts to produce even less of the hormone. Serotonin is converted into NAS (N-acetylserotonin) by AANAT (arylalkylamine N-acetyltransferase, EC: 2.3. 1.87), then converted into melatonin by ASMT (acetylserotonin N-methyltransferase. So if you have problems with methylation you would have problems with converting serotonin to melatonin. To fix this you'd need to make sure that you're not deficient in b12. If you're functionally deficient in vit. B2 you would get what's called 'paradoxical' b12 deficiency in which serum b12 is in normal to high range but mostly in inactive form. To fix this you first need to fix the functional B2 deficiency and for that you'd need to check which b2 cofactors you're deficient (Iodine, Selenium and Malybdenum)
You can't fix paradoxical b12 before fixing the functional B2 deficiency first.
Furthermore,
There are over 200 enzymes in the body involved in methylation
The major methyl donor for methylation is S-Adenosyl-Methionine (SAM).
Maintenance of methylation is critically dependent upon MethylCo(III)B12 and the enzyme methionine synthase (MTR)
The methylation cycle is also dependent upon two enzymes that require functional B2 sufficiency, MTHFR and MTRR
Maintenance of functional vitamin B12 requires functional B2 deficiency,
Lack of functional B2 in leads to functional B12 deficiency and a reduction in methylation activities
Functional Vitamin B2 deficiency can lead to functional B6 deficiency, exacerbating functional B12 deficiency
Functional Vitamin B2 deficiency and associated functional B12 deficiency, can lead to functional folate deficiency.
Reduced methylation has been associated with many conditions including dementia, Parkinson's disease, Autism, Chronic Fatigue Syndrome.
Inactivation of histamine by the enzyme Histamine-N-Methyl Transferase. Histamine-[HNMT] + SAM => N-methylhistamine + SAH. Deficiency in SAM leads to histamine intolerance and in extreme cases can lead to a condition mistakenly named Mast Cell Activation Syndrome.
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